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Three newly published peer-reviewed studies confirm that infants exposed
to mercury in utero suffer serious harm--including death.
1. A study in the journal, Human & Experimental Toxicology (Oct.
2012) examined FDA's Vaccine Adverse Event Reporting System (VAERS)
database and found an increase in reports of spontaneous abortion and
stillbirths following pregnant woman's vaccination against the flu and H1N1.
2. A report in the journal Toxicological & Environmental Chemistry (2012) found that the data "demonstrated that
Hg [mercury] exposures, particularly during the first trimester of pregnancy, at
well-established dose/weight ratios produced severe damage to humans
including death."
3. A third report in the journal Archives of Pediatric & Adolescent Medicine (a JAMA Network journal)
reports that prenatal exposure to mercury and fish is linked to
behavior associated with Attention Deficit Hypertension Disorder.
The evidence is clear that mercury is toxic for children's neurological development.
Although the government-industrial vaccine complex never acknowledged
the harm produced by mercury (Thimerisol) toxicity in children's
vaccines, it was removed from most childhood vaccines.
It should not come as a surprise that mercury is harmful to developing human
embryos--whether the mercury exposure is from fish, water, or vaccine
adjuvants.
What then are officials at the Centers for Disease Control thinking when
they recommend the flu vaccine--that contains mercury--for pregnant women?
Given that the flu is hardly a life-threatening illness, and given that
the evidence of efficacy for the flu vaccine has been found to be
negligible--at best--as determined by the Cochrane review, we question the morally irresponsible, medically unsupportable CDC
recommendation urging pregnant women to get vaccinated with a vaccine
containing mercury.
CDC officials who formulated the flu vaccine policy urging pregnant women
to be vaccinated with a vaccine containing mercury--in complete
disregard of the scientific evidence showing profound harm for the
unborn--should be fired and the recommendation rescinded.
Vera Sharav
Centers for Disease Control recommendation for pregnant women: cdc_recommendation-_flu_-_pregnant_women
Pregnant Women & Influenza (Flu)
Flu is more likely to cause severe illness in pregnant
women than in women who are not pregnant. Changes in the immune system,
heart, and lungs during pregnancy make pregnant women more prone to
severe illness from flu as well as hospitalizations and even death.
Pregnant woman with flu also have a greater chance for serious problems
for their unborn baby, including premature labor and delivery.
Flu shots will protect pregnant women, their unborn babies and even protect the baby after birth.
The Flu Shot is the Best Protection Against Flu
Getting
a flu shot is the first and most important step in protecting against
flu. The flu shot given during pregnancy has been shown to protect both
the mother and her baby (up to 6 months old) from flu. (The nasal spray
vaccine should not be given to women who are pregnant.) Learn more about
the flu vaccine.
The Flu Shot is Safe for Pregnant Women
Flu
shots are a safe way to protect the mother and her unborn child from
serious illness and complications of flu. The flu shot has been given to
millions of pregnant women over many years. Flu shots have not been
shown to cause harm to pregnant women or their babies. It is very
important for pregnant women to get the flu shot.
http://het.sagepub.com/content/early/2012/09/12/0960327112455067.abstract
1. GS Goldman.
Comparison of VAERS fetal-loss reports during three consecutive
influenza seasons,
Human & Experimental Toxicology, October, 2012
Was there a synergistic fetal toxicity associated with the two-vaccine 2009/2010 season?
Gary S Goldman, Independent Computer Scientist, P.O. Box 847, Pearblossom, CA 93553, USA Email:
This e-mail address is being protected from spam bots, you need JavaScript enabled to view it
Abstract
The aim of this study was to compare the
number of inactivated-influenza vaccine–related spontaneous abortion and
stillbirth
(SB) reports in the Vaccine Adverse Event Reporting
System (VAERS) database during three consecutive flu seasons beginning
2008/2009 and assess the relative fetal death
reports associated with the two-vaccine 2009/2010 season. The VAERS
database
was searched for reports of fetal demise following
administration of the influenza vaccine/vaccines to pregnant women.
Utilization
of an independent surveillance survey and VAERS,
two-source capture–recapture analysis estimated the reporting
completeness
in the 2009/2010 flu season. Capture–recapture
demonstrated that the VAERS database captured about 13.2% of the total
1321
(95% confidence interval (CI): 815–2795) estimated
reports, yielding an ascertainment-corrected rate of 590 fetal-loss
reports
per million pregnant women vaccinated (or 1 per
1695). The unadjusted fetal-loss report rates for the three consecutive
influenza
seasons beginning 2008/2009 were 6.8 (95% CI:
0.1–13.1), 77.8 (95% CI: 66.3–89.4), and 12.6 (95% CI: 7.2–18.0) cases
per million
pregnant women vaccinated, respectively. The
observed reporting bias was too low to explain the magnitude increase in
fetal-demise
reporting rates in the VAERS database relative to
the reported annual trends. Thus, a synergistic fetal toxicity likely
resulted
from the administration of both the pandemic
(A-H1N1) and seasonal influenza vaccines during the 2009/2010 season.
~~~~~~~~~~~~~~~~~~~~
http://www.tandfonline.com/doi/abs/10.1080/02772248.2012.724574
2. Ian A. Brown & David W. Austin.
Maternal transfer of mercury to the developing embryo/fetus: is there a safe level?
Toxicological & Environmental Chemistry Vol 94
2012
Abstract
Mercury
(Hg) exposure is ubiquitous in modern society via vaccines,
fish/crustacea, dental amalgam, food, water, and the atmosphere. This
article examines Hg exposure in the context of primary exposure to
pregnant women and secondary exposure experienced by their unborn
babies. Babies in utero are particularly at risk of higher Hg
exposure than adults (on a dose/weight basis through maternal Hg
transfer via the placenta), and are more susceptible to adverse effects
from mercury and its biologically active compounds. It is, therefore,
critical that regulatory advisories around maximum safe Hg exposures
account for pregnant women and secondary exposure that children in utero
experience. This study focused on standardized embryonic and fetal Hg
exposures via primary exposure to the pregnant mother of two common Hg
sources (dietary fish and parenteral vaccines). Data demonstrated that
Hg exposures, particularly during the first trimester of pregnancy, at
well-established dose/weight ratios produced severe damage to humans
including death.
In light of research suggestive of a mercuric risk
factor for childhood conditions such as tic disorders, cerebral palsy,
and autism, it is essential that Hg advisories account for secondary
prenatal human exposures.
~~~~~~~~~~~~~~~
http://archpedi.jamanetwork.com/article.aspx?articleid=1377487
3. Sharon K.
Sagiv, PhD, MPH; Sally W. Thurston, PhD; David C. Bellinger, PhD, MS;
Chitra Amarasiriwardena, PhD; Susan A. Korrick, MD, MPH
Prenatal
Exposure to Mercury and Fish Consumption During Pregnancy and
Attention-Deficit/Hyperactivity Disorder–Related Behavior in Children,
Archives of Pediatrics & Adolescent Medicine [JAMA Network] October,
2012.
ABSTRACT
Objective
To investigate the association of prenatal mercury
exposure and fish intake with attention-deficit/hyperactivity disorder
(ADHD)–related behavior.
Methods
For a population-based prospective birth cohort
recruited in New Bedford, Massachusetts (1993-1998), we analyzed data
for children examined at age 8 years with peripartum maternal hair
mercury measures (n = 421) or maternal report of fish consumption during
pregnancy (n = 515). Inattentive and impulsive/hyperactive behaviors
were assessed using a teacher rating scale and neuropsychological
testing.
Results
The median maternal hair mercury level was 0.45 μg/g
(range, 0.03-5.14 μg/g), and 52% of mothers consumed more than 2 fish
servings weekly. In multivariable regression models, mercury exposure
was associated with inattention and impulsivity/hyperactivity; some
outcomes had an apparent threshold with associations at 1 μg/g or
greater of mercury. For example, at 1 μg/g or greater, the adjusted risk
ratios for mild/markedly atypical inattentive and impulsive/hyperactive
behaviors were 1.4 (95% CI, 1.0-1.8) and 1.7 (95% CI, 1.2-2.4),
respectively, for an interquartile range (0.5 μg/g) mercury increase;
there was no confounding by fish consumption. For neuropsychological
assessments, mercury and behavior associations were detected primarily
for boys. There was a protective association for fish consumption (>2
servings per week) with ADHD-related behaviors, particularly
impulsive/hyperactive behaviors (relative risk = 0.4; 95% CI, 0.2-0.6).
Conclusions
Low-level prenatal mercury exposure is associated with
a greater risk of ADHD-related behaviors, and fish consumption during
pregnancy is protective of these behaviors. These findings underscore
the difficulties of balancing the benefits of fish intake with the
detriments of low-level mercury exposure in developing dietary
recommendations in pregnancy.
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